Inducen-MLD2

Induced Native Phage Therapy (INPT), harnesses known mechanisms of endogenous (native) bacteriophages—beneficial and benevolent viruses (phages) that naturally infect bacteria and often enter the human body already residing within bacteria, or otherwise already reside within the phageome of the body. This is accomplished by using technology called, Biospectral Emission Sequencing (BES) to isolate specific electromagnetic signatures. These signatures are imprinted into an oral liquid formula, called Inducen® formulas, which, when ingested, induces epigenetic changes in the quiescent (lysogenic) phages, prompting them to enter a lytic cycle where they actively replicate within and destroy targeted bacteria, including those in biofilms, without harming beneficial microbiota or causing adverse reactions, as demonstrated in clinical studies achieving up to 100% apparent pathogen clearance. Inducen formulations are believed to be essentially undetectable by the human body and therefore are naturally non-allergenic and are also completely non-toxic due to having no chemical component within the formula. The Inducen formulas contain no phages, working instead by activating specific populations of native phages to kill the targeted infectious agents. 

Aspergillus carneus, Aspergillus clavitus, Aspergillus flavus, Aspergillus glaucus, Aspergillus niger, Aspergillus ochraceus, Aspergillus nidulans, Aureobasilium pullulans, Aspergillus ustus, Aspergillus versicolor, Blastomyces dermatitis, Candida auris, Cephalospor, Chaetomium globosum, Cladosporium mix, Cryptococcus neoformans, Cylindrocarpon, Epicoccum mix, Fusarium mix, Geotrichum candidum, Glomus mosseae, FL1953), Glomus intraradices, Helminthosporum, Histoplasmosis, Memnoniella, Microsporum, Monilla albicans, Monotospora, Mixed Mucor, Mucor racemosus, Myothecium, , Mixed Penicillium, Penicillium aurantiogriseum, Penicillium chrysogenum, Penicillium nordicum, Penicillium verrucosum, Phoma mix, Pithomyces, Pullularia, Protomyxzoa rheumatica (aka: funneliformis mosseae, Rhizophus rubra, Rhizophus stolonifer, Rhodotorulla, Scopulariopsis, Smuts (barley, wheat, oats, grass), Spondylocladium, Sporothrix, Sportrichum pruinosum, Sporobolomyces, Stachybotrys, Stemphylium, Trichoderma mix, Wallemia mix, All associated Mycotoxins:  3-Acetyldeoxynivalenol (Fusarium), Acetyl T-2 toxin (Fusarium), Aflatoxin B1 (Aspergillus), Aflatoxin B2 (Aspergillus), Aflatoxin G1 (Aspergillus), Aflatoxin G2 (Aspergillus), Aflatoxin M1 (Aspergillus), Citreoviridin (Penicillium), Citrinin (Penicillium), Cyclopiazonic acid (Penicillium/Aspergillus), Cytochalasin B (Helminthosporium), Deoxynivalenol (Fusarium), Diacetoxyscirpenol (Fusarium), Fumonisin (Fusarium), Gliotoxin (Gliocladium/Aspergillus), Griseofulvin, Moniliformin (Fusarium), 3-Nitropropionic acid (multiple species), Ochratoxin A (Aspergillus/Penicillium species), Patulin (Aspergillus/Penicillium/Byssochlamys), Penetrem A (Penicillium crustosum), Radicinin (Alternaria), Roridin A (Stachybotrys), Rubratoxin (Penicillium rubrum/purpurrescens), Sterigmatocystin (Aspergillus nidulans/A. versicolor), Trichothecenes (Fusarium/Stachybotrys), T-2 toxin (Fusarium), HT-2 toxin (Fusarium species), T-2 tetraol (Fusarium), T-2 triol (Fusarium), Verrucarin A (Fusarium/Stachybotrys), Verrucarol (Fusarium), Zearalenone (Fusarium).

High-Impact (Test First)

• Aflatoxin B1 → Hepatocellular carcinoma
• Ochratoxin A → Chronic kidney disease
• Fumonisin B1 → Esophageal cancer, neural defects
• T-2 / HT-2 toxins → GI necrosis, marrow injury
• Macrocyclic trichothecenes → Neurotoxicity, inflammation

Moderate Impact

• Deoxynivalenol (vomiting, immune effects)
• Zearalenone (endocrine disruption)
• Sterigmatocystin (carcinogenic precursor)
• Citrinin (renal injury)

If labs + symptoms don’t point to liver, kidney, marrow, CNS, or severe infection — don’t over-interpret environmental mold panels. Focus on toxin burden + organ system, not species lists.

Strong evidence, reproducible human illness, systemic effects

Dominant Fungi

Aspergillus flavus

• Primary toxins: Aflatoxin B1, B2, G1, G2, M1
• Key conditions:
  • Hepatocellular carcinoma (Group 1 carcinogen)
  • Acute liver failure
  • Growth retardation, immune suppression
• Exposure: Food contamination, indoor mold

Aspergillus ochraceus / Penicillium verrucosum

• Primary toxin: Ochratoxin A
• Key conditions:
  • Chronic kidney disease
  • Balkan endemic nephropathy
  • Immunotoxicity, possible carcinogenicity

Stachybotrys (black mold)

• Primary toxins: Roridin A, Verrucarin A, macrocyclic trichothecenes
• Key conditions:
  • Severe inflammatory illness
  • Neurologic symptoms (headache, cognitive dysfunction)
  • Pulmonary hemorrhage (rare, controversial but documented)

Fusarium species (mix)

• Primary toxins: Fumonisins, DON, T-2, HT-2, Zearalenone
• Key conditions:
  • Esophageal cancer
  • Neural tube defects
  • Severe GI injury
  • Hematologic and immune suppression

Candida auris

• Not toxin-driven — infection-driven
• Key conditions:
  • Life-threatening fungemia
  • High mortality in ICU patients
  • Extreme drug resistance

Important Fungi

Aspergillus versicolor / A. nidulans

• Primary toxin: Sterigmatocystin (aflatoxin precursor)
• Key conditions:
  • Possible carcinogenicity
  • Chronic inflammatory symptoms
  • Indoor air exposure syndromes

Chaetomium globosum

• Primary toxins: Chaetoglobosins
• Key conditions:
  • Neurocognitive symptoms
  • Chronic inflammatory response
  • Co-exposure amplifier with Stachybotrys

Cryptococcus neoformans

• Key conditions:
  • Cryptococcal meningitis
  • Severe disease in HIV/transplant patients

Histoplasma capsulatum

• Key conditions:
  • Pulmonary histoplasmosis
  • Disseminated disease in immunocompromised

Blastomyces dermatitidis

• Key conditions:
  • Severe pneumonia
  • Bone, skin dissemination

Lower-Systemic but Still Relevant

Penicillium species

• Toxins: Citrinin, Patulin, Rubratoxin
• Conditions:
  • Nephrotoxicity (citrinin)
  • GI and immune irritation
  • Indoor mold illness

Cladosporium / Alternaria / Epicoccum

• Conditions:
  • Asthma
  • Allergic rhinitis
  • Hypersensitivity pneumonitis

Mucor / Rhizopus

• Conditions:
  • Mucormycosis (diabetics, transplant)
  • Rhino-orbital-cerebral disease (rare but lethal)

These are usually markers of exposure, not primary drivers of illness:

• Aureobasidium pullulans
• Wallemia
• Trichoderma
• Sporobolomyces
• Stemphylium
• Phoma
• Helminthosporium
• Smuts (grain exposure; mostly agricultural relevance)

Highest Concern

• Aflatoxin B1 → Liver cancer, acute hepatic failure
• Ochratoxin A → Kidney disease, immune suppression
• Fumonisin B1 → Neural tube defects, esophageal cancer
• T-2 / HT-2 toxins → Hematologic injury, GI necrosis
• Macrocyclic trichothecenes → Severe inflammation, neurotoxicity

Moderate Concern

• Deoxynivalenol (vomitoxin)
• Zearalenone (endocrine disruption)
• Sterigmatocystin
• Citrinin
• Patulin

Lower / Context-Dependent

• Cyclopiazonic acid
• Penetrem A
• Griseofulvin (mostly therapeutic relevance)
• Moniliformin

If you’re prioritizing real human illness, focus on:

1. Aspergillus (aflatoxin, ochratoxin, sterigmatocystin)
2. Fusarium (fumonisins, trichothecenes, zearalenone)
3. Stachybotrys (macrocyclic trichothecenes)
4. Candida auris (infection-driven mortality)
5. Histoplasma / Cryptococcus (true invasive mycoses)

*This presentation was largely generated by ChatGPT for educational purposes only, with no endorsement of PhagenCorp or the Inducen formulas implied or suggested. For licensed healthcare professional use and patient education.